The amount of compensatory sweating depends on the patient, the damage that the white rami communicans incurs, and the amount of cell body reorganization in the spinal cord after surgery.
Other potential complications include inadequate resection of the ganglia, gustatory sweating, pneumothorax, cardiac dysfunction, post-operative pain, and finally Horner’s syndrome secondary to resection of the stellate ganglion.
www.ubcmj.com/pdf/ubcmj_2_1_2010_24-29.pdf

After severing the cervical sympathetic trunk, the cells of the cervical sympathetic ganglion undergo transneuronic degeneration
After severing the sympathetic trunk, the cells of its origin undergo complete disintegration within a year.
http://onlinelibrary.wiley.com/doi/10.1111/j.1439-0442.1967.tb00255.x/abstract

Wednesday, June 24, 2009

Interview with Dr Telaranta

Do you find ETS patients less satisfied with their decision over time due
to CS or other factors & have there been adequate studies on the long term
effects of ETS on cardiac, skin function, etc..?

-My opinion is, that during time the past slowly gets into oblivion, the patients don’t remember any more the original situation. Also, I believe that the first hype of enthusiasm goes away with the central nervous system getting used to the new situation. It may even be that the initial effect on the thalamic structures is some kind of euphoric hybris due to the sudden increase of the serotonine-like overflow.

http://www.angelfire.com/journal2/sadhelp/Interview1.htm

Science 3 September 1971:
Vol. 173. no. 4000, pp. 931 - 934
DOI: 10.1126/science.173.4000.931
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Serum Dopamine-beta-Hydroxylase: Decrease after Chemical Sympathectomy

Richard Weinshilboum 1 and Julius Axelrod 2

1 Pharmacology-Toxicology Program, National Institute of General Medical Sciences, Bethesda, Maryland 20014
2 Laboratory of Clinical Science, National Institute of Mental Health, Bethesda, Maryland 20014

Dopamine-beta- hydroxylase is an enzyme that is localized to catecholamine-containing vesicles in sympathetic nerves and the adrenal medulla, and is also found in the serum. Treatment of rats with 6-hydroxydopamine, a drug which destroys sympathetic nerve terminals, leads to a decrease in serum dopamine-beta-hydroxylase activity. The decrease is not due to an effect on the adrenal medulla or to an increase in circulating inhibitor or inhibitors of enzyme. These data represent evidence that at least a portion of the circulating dopamine-beta-hydroxylase activity arises from sympathetic nerve terminals.